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We present a detection of 21 cm emission from large-scale structure (LSS) between redshift 0.78 and 1.43 made with the Canadian Hydrogen Intensity Mapping Experiment. Radio observations acquired over 102 nights are used to construct maps that are foreground filtered and stacked on the angular and spectral locations of luminous red galaxies (LRGs), emission-line galaxies (ELGs), and quasars (QSOs) from the eBOSS clustering catalogs. We find decisive evidence for a detection when stacking on all three tracers of LSS, with the logarithm of the Bayes factor equal to 18.9 (LRG), 10.8 (ELG), and 56.3 (QSO). An alternative frequentist interpretation, based on the likelihood ratio test, yields a detection significance of 7.1σ(LRG), 5.7σ(ELG), and 11.1σ(QSO). These are the first 21 cm intensity mapping measurements made with an interferometer. We constrain the effective clustering amplitude of neutral hydrogen (Hi), defined as${\mathit{}}_{\mathrm{H}\mathrm{I}}\equiv {10}^3{\mathrm{\Omega }}_{\mathrm{H}\mathrm{I}}\left(b_{\mathrm{H}\mathrm{I}}+〈f{\mu }^2〉\right)$, where Ω_Hiis the cosmic abundance of Hi,b_Hiis the linear bias of Hi, and 〈fμ²〉 = 0.552 encodes the effect of redshift-space distortions at linear order. We find${\mathit{}}_{\mathrm{H}\mathrm{I}}={1.51}_{-0.97}^{+3.60}$for LRGs (z= 0.84),${\mathit{}}_{\mathrm{H}\mathrm{I}}={6.76}_{-3.79}^{+9.04}$for ELGs (z= 0.96), and${\mathit{}}_{\mathrm{H}\mathrm{I}}={1.68}_{-0.67}^{+1.10}$for QSOs (z= 1.20), with constraints limited by modeling uncertainties at nonlinear scales. We are also sensitive to bias in the spectroscopic redshifts of each tracer, and we find a nonzero bias Δv= − 66 ± 20 km s⁻¹for the QSOs. We split the QSO catalog into three redshift bins and have a decisive detection in each, with the upper bin atz= 1.30 producing the highest-redshift 21 cm intensity mapping measurement thus far.

COVID‐19 has highlighted a brutal reality known for decades, that Black, Indigenous, and People of Color bear a disproportionate burden of US annual sepsis cases. While plentiful research funds have been spent investigating genetic reasons for racial disparities in sepsis, an abundance of research shows that sepsis incidence and mortality maps to indicators of colonial practices including residential segregation, economic and marginalization sepsis, and denial of care. Here we argue that sepsis risk is an immunological embodiment of racism in colonial states, that the factors contributing to sepsis disparities are insidious and systemic. We show that regardless of causative pathogen, or host ancestry, racialized people get and die of sepsis most frequently in a pattern repeatedly reiterated worldwide. Lastly, we argue that while alleviation of sepsis disparities requires radical, multiscale intervention, biological anthropologists have a responsibility in this crisis. While some of us can harness our expertise to take on the ground action in sepsis prevention, all of us can leverage our positions as the first point of contact for in depth human biology instruction on most college campuses. As a leading cause of death worldwide, and a syndrome that exhibits the interplay between human physiology, race and environment, sepsis is at the nexus of major themes in biological anthropology and is a natural fit for the field's curriculum. In adopting a discussion of race and sepsis in our courses, we not only develop new research areas but increase public awareness of both sepsis and the factors contributing to uneven sepsis burden.

Rising sea levels, subsidence, and decreased fluvial sediment load threaten river deltas and their wetlands. However, the feedbacks between fluvial and non‐fluvial (marsh) deposition remain weakly constrained. We investigate how non‐riverine, elevation‐controlled deposition typified by marshes impacts sediment partitioning between a delta's topset, coastal zone, and foreset by comparing a delta experiment with proxy marsh accumulation to a control. Marsh accumulation alters fluvial sediment distribution by decreasing the slope in the marsh window by ∼50%, creating a 78% larger marsh zone. Fluvial incursions into the marsh window trap 1.3 times more clastic volume. The volume exported to deep water remains unchanged. Marsh deposition shifts elevation distributions toward sea level, which produces a hypsometry akin to field‐scale deltas. The elevation‐lowering effect of marshes on an equilibrium delta shown here constitutes an unexplored feedback and an important aspect of coastal sustainability.